Peer-Reviewed German Study: Direct Evidence People Died from mRNA-Vaccine-Damaged Hearts

In this video analyzing the results of the German study, Dr. John Campbell, who is based in the United Kingdom, told his audience: “This is peer-reviewed. This is proper science, and a definitive pathological diagnosis by a group of leading German pathologists.”

Original video: Myocarditis German evidence - https://youtu.be/j_DdSMn55cA

People Died from mRNA-Vaccine-Damaged Hearts, New Peer-Reviewed German Study Provides Direct Evidence
https://www.globalresearch.ca/people-died-from-mrna-vaccine-damaged-hearts-new-peer-reviewed-german-study-provides-direct-evidence/5802451

Medical pathologists from Heidelberg University Hospital in Heidelberg, Germany have published direct evidence showing how people found dead after mRNA vaccination died. As this team of six scientists explore in their study, these mRNA-vaccinated patients suffered from heart damage because their hearts were attacked by their own immune cells. This autoimmune attack on their own heart cells then leads to their damaged hearts beating so many times per second that, once the tachycardia unexpectedly started, they died in minutes.

The article, “Autopsy-based histopathological characterization of myocarditis after anti-SARS-CoV-2-vaccination,” was published on Nov. 27, 2022, in the journal Clinical Research in Cardiology, the official journal of the German Cardiac Society. The research team autopsied 25 victims of different ages who were found dead at home within 28 days of vaccination. They looked at their heart tissue under the microscope to find out why these people died of cardiac rhythmic disruption when they had no apparent underlying heart disease.

In the authors’ own words: “Our findings establish the histological phenotype of lethal vaccination-associated myocarditis.”

Histological phenotype means direct observation of microscopic tissue.

Autopsy-based histopathological characterization of myocarditis after anti-SARS-CoV-2-vaccination
https://link.springer.com/article/10.1007/s00392-022-02129-5

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